Clinical Studies of Non-B Posttransfusion Hepatitis
نویسندگان
چکیده
منابع مشابه
Serum hepatitis C virus sequences in posttransfusion non-A, non-B hepatitis.
We investigated 17 patients (12 males and 5 females, ages 2 to 57 years old) with posttransfusion non-A, non-B hepatitis to determine relationships between clinical courses and hepatitis C virus (HCV) markers. The patients were grouped according to time course of abnormal serum alanine aminotransferase (ALT) levels into three categories (chronic biochemical disease, biochemically resolved chron...
متن کاملSTUDIES ON HEPATITIS B VACCINES
HBs vaccines were prepared by three different methods: 1) Heat inactivation (Krugman, et.al 1971)2) Ammonium sulphate precipitation followed by column chromatography (Sephadex G-200) 3) PEG-6000 (polyethylene glycol) precipitation followed by column chromatography using Sephadex G-200. Their efficacy was studied in guinea pigs, and the results compared with commercial Japanese vaccine (Gr...
متن کاملHistory of posttransfusion hepatitis.
The risk of hepatitis virus transmission from tranfusions has declined dramatically from that of the 1940s when posttransfusion hepatitis (PTH) was first appreciated. Introduction of hepatitis B surface antigen screening and conversion to volunteer donors for whole-blood donations in the late 1960s and early 1970s led to substantial reduction in PTH cases. However, up to 10% of the recipients c...
متن کاملPosttransfusion hepatitis after exclusion of commercial and hepatitis-B antigen-positive donors.
In a prospective study the exclusion of commercial blood donors and donors positive for hepatitis-B antigen (HBAg) resulted in a hepatitis frequency of only 3 .7 cases/1000 units transfused. Residual posttransfusion hepatitis was caused by at least two viruses. Four of 10 cases were short incubation, HBAg negative; six cases were long incubation, HBAg positive. One patient had both diseases in ...
متن کاملStudies on the pathophysiology of posttransfusion purpura.
Posttransfusion purpura typically occurs in PLA1 negative blood recipients who have been previously immunized to the PLA1 antigen. Following transfusion, severe thrombocytopenia develops with the formation of anti-PLA1. Since the patients' platelets lack the PLA1 antigen, one would not expect this antibody to destroy autologous platelets. In this study we show that PLA1 antigen exists in stored...
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ژورنال
عنوان ژورنال: Kanzo
سال: 1978
ISSN: 0451-4203,1881-3593
DOI: 10.2957/kanzo.19.725